Colorectal cancer

A new cellular model of colorectal cancer helps identify high-risk subtypes likely to resist standard treatments and proliferate in response.

Region-specific stem cells may not only act as starting points for colorectal cancer, but also offer new clues for early detection and treatment.

High iron levels, often linked to red meat consumption, can fuel cancer growth by reactivating proteins that allow cancer cells to divide endlessly.

Researchers have discovered a molecular pathway in colorectal cancer cells that leads to uncontrolled division, creating an avenue for more targeted therapies.

Different forms of the protein HNF4ɑ can pair up to influence diverse processes ranging from development to diabetes, study shows.

Thermal stress elicits genomic changes that can make cancer cells resistant to chemotherapy

A
popular diabetes drug could one day be used to treat malignant tumors

Analyzing gene activity in single cells offers a
clearer view of bowel cancer

A surprising finding shows that some clusters of cells commonly found in the bloodstream of colorectal cancer patients are actually non-cancerous

Trapping cells that tumors release into the bloodstream allows fast and sensitive genetic analysis

A protein originally identified as a tumor suppressor also helps establish body patterns in developing embryos