New insights into the age-associated changes affecting macrophages offer promising avenues for the treatment of chronic wounds.
Researchers unravel a complex inflammatory pathway involved in severe COVID-19 infection and identify a therapeutic mechanism to reverse it.
Elevated levels of inflammatory factors may be linked to persistent COVID-19 symptoms, researchers find.
In COVID-19 long-haulers, an unrelenting immune response damages blood vessels, making them more susceptible to developing blood clots.
The mysterious trigger of the NLRP1 inflammasome sensor has finally been identified: a protease found in the common cold-causing human rhinovirus tells our body that it has been invaded.
A*STAR researchers identify a novel mechanism in which an inflammatory protein heals wounds in healthy individuals but has the opposite effect in diabetics.
Controlling the delicate balance between immune cells called granulocytes and monocytes could make the difference between a healthy immune response and life-threatening sepsis.
In a classic case of double trouble, the binding of SARS-CoV-2’s spike protein to bacterial lipopolysaccharide supercharges inflammation in COVID-19 patients.
These novel immune signatures identify patients most at risk for developing severe COVID-19 symptoms.
Lisa F. P. Ng, a leading infectious disease expert on infection and immunity, shares how her team’s prior experience has helped them quickly understand the nature of SARS-CoV-2, the virus that causes COVID-19.
A*STAR scientists can now classify immune cell populations with greater precision thanks to a machine-learning algorithm.